Search Result

Results for "

miR-16

" in MedChemExpress (MCE) Product Catalog:

By Targets:	Apoptosis (3) MicroRNA (23) Protein Arginine Deiminase (3) Small Interfering RNA (siRNA) (3)
By Research Areas:	Cancer (23) Inflammation/Immunology (3)
Oligonucleotides:	miRNA mimics (5) miRNA agomirs (5) miRNA antagomirs (5) MicroRNAs (20) Rat Pre-designed siRNA Sets (1) miRNA inhibitors (5) Mouse Pre-designed siRNA Sets (1) Human Pre-designed siRNA Sets (1) siRNAs (3)

Inhibitors & Agonists

Oligonucleotides

Targets Recommended: Human leukocyte immunoglobulin (Ig)-like receptors (LILR)

Cat. No.	Product Name	Target	Research Areas	Chemical Structure

HY-100574A

Cl-amidine hydrochloride Maximum Cited Publications 15 Publications Verification	Protein Arginine Deiminase Apoptosis MicroRNA	Inflammation/Immunology Cancer
Cl-amidine hydrochloride is an orally active peptidylarginine deminase (PAD) inhibitor, with IC₅₀ values of 0.8 μM, 6.2 μM and 5.9 μM for PAD1, PAD3, and PAD4, respectively. Cl-amidine hydrochloride induces apoptosis in cancer cells. Cl-amidine hydrochloride induces microRNA (miR)-16 (miRNA-16, microRNA-16) expression and causes cell cycle arrest. Cl-Amidine hydrochloride prevents histone 3 citrullination and neutrophil extracellular trap formation, and improves survival in a murine sepsis model .

HY-100574

Cl-amidine	Protein Arginine Deiminase Apoptosis MicroRNA	Inflammation/Immunology Cancer
Cl-amidine is an orally active peptidylarginine deminase (PAD) inhibitor, with IC₅₀ values of 0.8 μM, 6.2 μM and 5.9 μM for PAD1, PAD3, and PAD4, respectively. Cl-amidine induces apoptosis in cancer cells. Cl-amidine induces microRNA (miR)-16 (miRNA-16, microRNA-16) expression and causes cell cycle arrest. Cl-Amidine prevents histone 3 citrullination and neutrophil extracellular trap formation, and improves survival in a murine sepsis model .

HY-100574B

Cl-amidine TFA	Protein Arginine Deiminase Apoptosis MicroRNA	Inflammation/Immunology Cancer
Cl-amidine TFA is an orally active peptidylarginine deminase (PAD) inhibitor, with IC₅₀ values of 0.8 μM, 6.2 μM and 5.9 μM for PAD1, PAD3, and PAD4, respectively. Cl-amidine TFA induces apoptosis in cancer cells. Cl-amidine TFA induces microRNA (miR)-16 (miRNA-16, microRNA-16) expression and causes cell cycle arrest. Cl-Amidine TFA prevents histone 3 citrullination and neutrophil extracellular trap formation, and improves survival in a murine sepsis model .

HY-R04248

*rno-miR-16-3p mimic*	MicroRNA	Cancer
rno-miR-16-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.

rno-miR-16-3p mimic rno-miR-16-3p mimic

HY-R00324

*hsa-miR-16-5p mimic*	MicroRNA	Cancer
hsa-miR-16-5p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.

hsa-miR-16-5p mimic hsa-miR-16-5p mimic

HY-R02693

*mmu-miR-16-1-3p mimic*	MicroRNA	Cancer
mmu-miR-16-1-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.

mmu-miR-16-1-3p mimic mmu-miR-16-1-3p mimic

HY-R00323

*hsa-miR-16-2-3p mimic*	MicroRNA	Cancer
hsa-miR-16-2-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.

hsa-miR-16-2-3p mimic hsa-miR-16-2-3p mimic

HY-R00322

*hsa-miR-16-1-3p mimic*	MicroRNA	Cancer
hsa-miR-16-1-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.

hsa-miR-16-1-3p mimic hsa-miR-16-1-3p mimic

HY-RI00324

*hsa-miR-16-5p inhibitor*	MicroRNA	Cancer
hsa-miR-16-5p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.

hsa-miR-16-5p inhibitor hsa-miR-16-5p inhibitor

HY-RI04248

*rno-miR-16-3p inhibitor*	MicroRNA	Cancer
rno-miR-16-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.

rno-miR-16-3p inhibitor rno-miR-16-3p inhibitor

HY-RI00323

*hsa-miR-16-2-3p inhibitor*	MicroRNA	Cancer
hsa-miR-16-2-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.

hsa-miR-16-2-3p inhibitor hsa-miR-16-2-3p inhibitor

HY-RI02693

*mmu-miR-16-1-3p inhibitor*	MicroRNA	Cancer
mmu-miR-16-1-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.

mmu-miR-16-1-3p inhibitor mmu-miR-16-1-3p inhibitor

HY-RI00322

*hsa-miR-16-1-3p inhibitor*	MicroRNA	Cancer
hsa-miR-16-1-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.

hsa-miR-16-1-3p inhibitor hsa-miR-16-1-3p inhibitor

HY-R00324A

*hsa-miR-16-5p agomir*	MicroRNA	Cancer
hsa-miR-16-5p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

hsa-miR-16-5p agomir hsa-miR-16-5p agomir

HY-R04248A

*rno-miR-16-3p agomir*	MicroRNA	Cancer
rno-miR-16-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

rno-miR-16-3p agomir rno-miR-16-3p agomir

HY-R02693A

*mmu-miR-16-1-3p agomir*	MicroRNA	Cancer
mmu-miR-16-1-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

mmu-miR-16-1-3p agomir mmu-miR-16-1-3p agomir

HY-R00323A

*hsa-miR-16-2-3p agomir*	MicroRNA	Cancer
hsa-miR-16-2-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

hsa-miR-16-2-3p agomir hsa-miR-16-2-3p agomir

HY-R00322A

*hsa-miR-16-1-3p agomir*	MicroRNA	Cancer
hsa-miR-16-1-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

hsa-miR-16-1-3p agomir hsa-miR-16-1-3p agomir

HY-RI00324A

*hsa-miR-16-5p antagomir*	MicroRNA	Cancer
hsa-miR-16-5p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

hsa-miR-16-5p antagomir hsa-miR-16-5p antagomir

HY-RI04248A

*rno-miR-16-3p antagomir*	MicroRNA	Cancer
rno-miR-16-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

rno-miR-16-3p antagomir rno-miR-16-3p antagomir

HY-RI00322A

*hsa-miR-16-1-3p antagomir*	MicroRNA	Cancer
hsa-miR-16-1-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

hsa-miR-16-1-3p antagomir hsa-miR-16-1-3p antagomir

HY-RI02693A

*mmu-miR-16-1-3p antagomir*	MicroRNA	Cancer
mmu-miR-16-1-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

mmu-miR-16-1-3p antagomir mmu-miR-16-1-3p antagomir

HY-RI00323A

*hsa-miR-16-2-3p antagomir*	MicroRNA	Cancer
hsa-miR-16-2-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.

hsa-miR-16-2-3p antagomir hsa-miR-16-2-3p antagomir

HY-RS27865

Gde1 Rat Pre-designed siRNA Set A	Small Interfering RNA (siRNA)	Others
Gde1 Rat Pre-designed siRNA Set A contains three designed siRNAs for Gde1 gene (Rat), as well as a negative control, a positive control, and a FAM-labeled negative control.

Gde1 Rat Pre-designed siRNA Set A Gde1 Rat Pre-designed siRNA Set A

HY-RS05348

GDE1 Human Pre-designed siRNA Set A	Small Interfering RNA (siRNA)	Others
GDE1 Human Pre-designed siRNA Set A contains three designed siRNAs for GDE1 gene (Human), as well as a negative control, a positive control, and a FAM-labeled negative control.

GDE1 Human Pre-designed siRNA Set A GDE1 Human Pre-designed siRNA Set A

HY-RS21346

Gde1 Mouse Pre-designed siRNA Set A	Small Interfering RNA (siRNA)	Others
Gde1 Mouse Pre-designed siRNA Set A contains three designed siRNAs for Gde1 gene (Mouse), as well as a negative control, a positive control, and a FAM-labeled negative control.

Gde1 Mouse Pre-designed siRNA Set A Gde1 Mouse Pre-designed siRNA Set A

Cat. No.	Product Name		Classification