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Results for "

atherosclerotic lesion

" in MedChemExpress (MCE) Product Catalog:

16

Inhibitors & Agonists

1

Biochemical Assay Reagents

3

Natural
Products

5

Isotope-Labeled Compounds

Cat. No. Product Name Target Research Areas Chemical Structure
  • HY-W010697
    Cholesteryl linoleate

    		Endogenous Metabolite Metabolic Disease
    Cholesteryl linoleate is shown to be the major cholesteryl ester contained in LDL and atherosclerotic lesions.
    Cholesteryl linoleate
  • HY-130587
    Sultosilic acid piperazine salt

    Piperazine sultosylate; A-585

    		 Others Metabolic Disease
    Sultosilic acid piperazine salt (Piperazine sultosylate; A-585) is a lipid-lowering agent. Sultosilic acid piperazine salt modifies the blood lipids levels, reduces platelet adhesiveness without promoting peroxisomal activity of hepatocytes or producing other adverse side-effects .
    Sultosilic acid piperazine salt
  • HY-W010697S
    Cholesteryl linoleate-d11

    		Isotope-Labeled Compounds Endogenous Metabolite Metabolic Disease
    Cholesteryl linoleate-d11 is deuterium labeled Cholesteryl linoleate. Cholesteryl linoleate is shown to be the major cholesteryl ester contained in LDL and atherosclerotic lesions .
    Cholesteryl linoleate-d11
  • HY-W013812S
    Ethyl linoleate-13C18

    Linoleic Acid-13C18 ethyl ester; Mandenol-13C18

    		 Isotope-Labeled Compounds Cardiovascular Disease
    Ethyl linoleate- 13C18 is the 13C labeled Ethyl linoleate. Ethyl linoleate inhibit the development of atherosclerotic lesions and the expression of inflammatory mediators .
    Ethyl linoleate-13C18
  • HY-N1553
    Pseudolaric acid D

    		Others Cardiovascular Disease Inflammation/Immunology
    Pseudolaric acid D is a compound with oral activity that can be isolated from Pseudolarix kaempferi Gorden. Pseudolaric acid D can significantly improve lipid metabolism, reduce atherosclerotic lesion area, and alleviate atherosclerotic changes in vascular wall. Pseudolaric acid D significantly inhibits the inflammatory reaction .
    Pseudolaric acid D
  • HY-W013812R
    Ethyl linoleate (Standard)

    		Others Cardiovascular Disease
    Ethyl linoleate (Standard) is the analytical standard of Ethyl linoleate. This product is intended for research and analytical applications. Ethyl linoleate (Linoleic Acid ethyl ester) inhibit the development of atherosclerotic lesions and the expression of inflammatory mediators .
    Ethyl linoleate (Standard)
  • HY-W013812S3
    Ethyl linoleate-d11

    Linoleic Acid ethyl ester-d11; Mandeno-d11

    		 Isotope-Labeled Compounds Others Cardiovascular Disease
    Ethyl linoleate-d11 (Linoleic Acid ethyl ester-d11) is deuterium labeled Ethyl linoleate. Ethyl linoleate (Linoleic Acid ethyl ester) inhibit the development of atherosclerotic lesions and the expression of inflammatory mediators .
    Ethyl linoleate-d11
  • HY-W013812S1
    Ethyl linoleate-d5

    Linoleic Acid ethyl ester-d5; Ethyl linoleate, pure-d5; Mandenol-d5

    		 Isotope-Labeled Compounds Others Cardiovascular Disease
    Ethyl linoleate-d5 (Linoleic Acid ethyl ester-d5) is deuterium labeled Ethyl linoleate. Ethyl linoleate (Linoleic Acid ethyl ester) inhibit the development of atherosclerotic lesions and the expression of inflammatory mediators .
    .
    Ethyl linoleate-d5
  • HY-W013812S2
    Ethyl linoleate-d2

    Linoleic Acid ethyl ester-d2; Mandenol-d2

    		 Isotope-Labeled Compounds Cardiovascular Disease
    Ethyl linoleate-d2 (Linoleic Acid ethyl ester-d2; Mandenol-d2) is the deuterium labeled Ethyl linoleate (HY-W013812). Ethyl linoleate (Linoleic Acid ethyl ester) inhibit the development of atherosclerotic lesions and the expression of inflammatory mediators .
    Ethyl linoleate-d2
  • HY-123016
    9(S)-HODE cholesteryl ester

    		Endogenous Metabolite Cardiovascular Disease
    9(S)-HODE cholesteryl ester is originally extracted from atherosclerotic lesions. It remains uncertain whether the oxidized fatty acid portion of the molecule results from enzymatic lipoxygenation or from random lipid peroxidation.2 9(S)-HODE cholesteryl ester can be used as a standard for analysis of chiral HODE cholesteryl esters.
    9(S)-HODE cholesteryl ester
  • HY-125394
    9(R)-HODE cholesteryl ester

    		Endogenous Metabolite Cardiovascular Disease
    9(R)-HODE cholesteryl ester is originally extracted from atherosclerotic lesions. It remains uncertain whether the oxidized fatty acid portion of the molecule results from enzymatic lipoxygenation or from random lipid peroxidation.2 9(R)-HODE cholesteryl ester can be used as a standard for analysis of chiral HODE cholesteryl esters.
    9(R)-HODE cholesteryl ester
  • HY-W747575
    (+/-)13-HODE cholesteryl ester

    		Endogenous Metabolite Metabolic Disease
    (±)13-HODE cholesteryl ester is originally extracted from atherosclerotic lesions1 and shown to be produced by Cu2+-catalyzed oxidation of LDL. Later studies determined that 15-LO from rabbit reticulocytes and human monocytes were able to metabolize cholesteryl linoleate, a major component of LDL, to 13-HODE cholesteryl ester.
    (+/-)13-HODE cholesteryl ester
  • HY-W424839
    Probucol dithiobisphenol

    DTBP

    		 Heme Oxygenase (HO) Cardiovascular Disease
    Probucol dithiobisphenol (DTBP) is the derivative of Probucol (HY-B0388). Probucol dithiobisphenol induces the production of heme oxygenase 1 (HO-1), and exhibits antioxidant activity. Probucol dithiobisphenol inhibits atherosclerotic lesions in Apoe −/− mice aorta, promotes re-endothelialization of aorta and inhibits restenosis in rabbit arterial injury models. Probucol dithiobisphenol is orally active .
    Probucol dithiobisphenol
  • HY-123016A
    (±)9-HODE cholesteryl ester

    		Endogenous Metabolite Metabolic Disease
    (±)9-HODE cholesteryl ester is originally extracted from atherosclerotic lesions1 and shown to be produced by Cu2+-catalyzed oxidation of LDL.2 Later studies determined that 15-LO from rabbit reticulocytes and human monocytes were able to metabolize cholesteryl linoleate, a major component of LDL, to 9-HODE cholesteryl ester.
    (±)9-HODE cholesteryl ester
  • HY-173234
    GI-Y2

    		Pyroptosis Cardiovascular Disease
    GI-Y2 is an inhibitor of GSDMD and can inhibit Pyroptosis of macrophages induced by oxidized low-density lipoprotein (ox-LDL). In vivo, GI-Y2 can dose-dependently reduce atherosclerotic plaques in ApoE-/- mice and decrease the lesion size and fibrosis degree of the aortic root. GI-Y2 is expected to be used in the research of pyroptosis and cardiovascular diseases, such as atherosclerosis .
    GI-Y2
  • HY-130502
    5β,6β-epoxycholestanol

    Cholesterol 5beta,6beta-epoxide

    		 Biochemical Assay Reagents Others
    5β,6β-epoxycholestanol is an oxidative metabolite of cholesterol formed by free-radical and non-radical oxidation of cholesterol at the 5,6 double bond. Induces lactate dehydrogenase (LDH) release and apoptosis in macrophage-differentiated U937 cells. Cholesterol 5beta,6beta-epoxide has been found in human fatty streaks and advanced atherosclerotic lesions, but not in normal aortic tissue .
    5β,6β-epoxycholestanol

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