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Results for "

Succinyl-CoA

" in MedChemExpress (MCE) Product Catalog:

9

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4

Natural
Products

1

Recombinant Proteins

Cat. No. Product Name Target Research Areas Chemical Structure
  • HY-137808
    Succinyl-Coenzyme A sodium
    1 Publications Verification

    Succinyl-CoA sodium

    Endogenous Metabolite Neurological Disease Metabolic Disease
    Succinyl-Coenzyme A (Succinyl-CoA) sodium is an intermediate of the citric acid cycle. Succinyl-Coenzyme A sodium can be converted to succinic acid and can also combines with glycine to form δ-ALA to synthesize porphyrins (heme). Succinyl-Coenzyme A sodium can be used in the study of metabolic, neurological and haematological abnormalities (such as porphyrias) caused by nutritional vitamin B12 deficiency (resulting in a deficiency in Succinyl-Coenzyme A synthesis) .
    Succinyl-Coenzyme A sodium
  • HY-126791A

    IsoSuccinyl coenzyme A tetralithium; Methylmalonyl coenzyme tetralithium

    Endogenous Metabolite Others
    Methylmalonyl-CoA (Methylmalonyl coenzyme A) tetralithium is a catabolite of valine, isoleucine, methionine, threonine, odd-chain fatty acids, and cholesterol. Methylmalonyl-CoA tetralithium is converted to succinyl-CoA by enzymatic reaction of methylmalonyl-CoA mutase (MCM) with coenzyme vitamin B12 .
    Methylmalonyl-CoA tetralithium
  • HY-148285

    Endogenous Metabolite Metabolic Disease
    Succinyl CoA is an intermediate of the citric acid cycle. Succinyl CoA can be converted to succinic acid and can also combines with glycine to form δ-amino levulinic acid (ALA) to synthesize porphyrins (heme). Succinyl CoA can be used in the study of metabolic, neurological and haematological abnormalities (such as porphyrias) caused by nutritional vitamin B12 deficiency (resulting in a deficiency in Succinyl CoA synthesis) .
    Succinyl CoA
  • HY-E70121

    Endogenous Metabolite Mitochondrial Metabolism Others
    Succinyl-CoA synthetase catalyzes the only substrate-level phosphoryl-ation step in the tricarboxylic acid cycle. Succinyl-CoA synthetase is a phosphate target for the activation of mitochondrial metabolism .
    Succinyl-CoA synthetase
  • HY-CE00712

    Naphthyl-2-methyl-Succinyl-CoA

    Biochemical Assay Reagents Metabolic Disease
    2-(2-Naphthylmethyl)succinyl-CoA (Naphthyl-2-methyl-succinyl-CoA) is a coenzyme A derivative.
    2-(2-Naphthylmethyl)succinyl-CoA
  • HY-126791

    IsoSuccinyl coenzyme A; Methylmalonyl coenzyme A

    Endogenous Metabolite Others
    Methylmalonyl coenzyme A (Methylmalonyl-CoA) is a catabolite of valine, isoleucine, methionine, threonine, odd-chain fatty acids, and cholesterol. Methylmalonyl coenzyme A tetralithium is converted to succinyl-CoA by enzymatic reaction of methylmalonyl-CoA mutase (MCM) with coenzyme vitamin B12 .
    Methylmalonyl-CoA
  • HY-CE00006

    (1-Methylpentyl)Succinyl-coenzyme A

    Biochemical Assay Reagents Others
    (1-Methylpentyl)succinyl-CoA ((1-Methylpentyl)succinyl-coenzyme A) is a a higher alkylsuccinyl-CoA .
    (1-Methylpentyl)succinyl-CoA
  • HY-CE00290

    (2S)-2-((R)-Hydroxybenzyl)Succinyl-coenzyme A

    Biochemical Assay Reagents Metabolic Disease
    (R,S)-(Hydroxy(phenyl)methyl)succinyl-CoA ((2S)-2-((R)-Hydroxybenzyl)succinyl-coenzyme A) is a [hydroxy(phenyl)methyl]succinyl-CoA.
    (R,S)-(Hydroxy(phenyl)methyl)succinyl-CoA
  • HY-173527

    Others Metabolic Disease
    PSSI-51 is an orally active, peripherally selective inhibitor of succinyl-CoA:3-ketoacid-CoA transferase (SCOT). PSSI-51 inhibits SCOT activity in peripheral tissues (such as muscle and kidney) but does not affect SCOT activity in brain tissue. PSSI-51 reduces ketone body oxidation by inhibiting SCOT, thereby improving obesity-related hyperglycemia. PSSI-51 can be used in the study of type 2 diabetes (T2D) and has the potential to improve obesity-related metabolic disorders .
    PSSI-51

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