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5-Methyl-2'-deoxycytidine

" in MedChemExpress (MCE) Product Catalog:

By Targets:	DNA Methyltransferase (1) DNA/RNA Synthesis (1) Drug Derivative (1) Endogenous Metabolite (1) Isotope-Labeled Compounds (1) Nucleoside Antimetabolite/Analog (1)
By Research Areas:	Cancer (2)
Oligonucleotides:	Nucleosides and their Analogs (2) C (2)

4

Inhibitors & Agonists

1

Natural
Products

1

Isotope-Labeled Compounds

2

Oligonucleotides

Cat. No.	Product Name	Target	Research Areas	Chemical Structure

**5-Methyl-2'-deoxycytidine** 3 Publications Verification 5-Methyldeoxycytidine	DNA Methyltransferase Endogenous Metabolite	Others
5-Methyl-2'-deoxycytidine (5mdC) is an endogenous substrate of DNA methyltransferases (such as mammalian 5-C-MTase) and binds to DNA dependent on the formation of DNA stem-loop structures. 5-Methyl-2'-deoxycytidine guides de novo DNA methylation by acting as a methylation mark and activates the methylation of adjacent CpG sites in single-stranded DNA through cis action. 5-Methyl-2'-deoxycytidine regulates DNA methylation patterns by recruiting methyltransferases to specific chromatin regions, affecting chromatin condensation and gene expression. Its distribution in plant cells is related to cell proliferation and differentiation stages. The methylation level of 5-Methyl-2'-deoxycytidine is low in proliferating cells and high in differentiated cells .

5-Methyl-2'-deoxycytidine-d3 5-Methyldeoxycytidine-d₃	Isotope-Labeled Compounds	Others
5-Methyl-2'-deoxycytidine-d3 is the deuterium labeled Methyl-2'-deoxycytidine .

5-Hydroxymethyl-2’-deoxycytidine 5hmdC	Drug Derivative DNA/RNA Synthesis	Cancer
5-Hydroxymethyl-2’-deoxycytidine (5hmdC) is an oxidation derivative of 5-methyl-2'-deoxycytidine (5-mdC) in DNA. 5-Hydroxymethyl-2’-deoxycytidine may serve as a marker of irreversibly damaged cells .

N4-Acetyl-5’-O-(4,4’-dimethoxytrityl)-5-methyl-2’-deoxycytidine	Nucleoside Antimetabolite/Analog	Cancer
N4-Acetyl-5’-O-(4,4’-dimethoxytrityl)-5-methyl-2’-deoxycytidine is a purine nucleoside analog. Purine nucleoside analogs have broad antitumor activity targeting indolent lymphoid malignancies. Anticancer mechanisms in this process rely on inhibition of DNA synthesis, induction of apoptosis, etc .

Cat. No.	Product Name	Category	Target	Chemical Structure

**5-Methyl-2'-deoxycytidine** 3 Publications Verification 5-Methyldeoxycytidine	Structural Classification Natural Products Classification of Application Fields Source classification Other Diseases Endogenous metabolite Disease Research Fields	DNA Methyltransferase Endogenous Metabolite
5-Methyl-2'-deoxycytidine (5mdC) is an endogenous substrate of DNA methyltransferases (such as mammalian 5-C-MTase) and binds to DNA dependent on the formation of DNA stem-loop structures. 5-Methyl-2'-deoxycytidine guides de novo DNA methylation by acting as a methylation mark and activates the methylation of adjacent CpG sites in single-stranded DNA through cis action. 5-Methyl-2'-deoxycytidine regulates DNA methylation patterns by recruiting methyltransferases to specific chromatin regions, affecting chromatin condensation and gene expression. Its distribution in plant cells is related to cell proliferation and differentiation stages. The methylation level of 5-Methyl-2'-deoxycytidine is low in proliferating cells and high in differentiated cells .

Cat. No.	Product Name	Chemical Structure

5-Methyl-2'-deoxycytidine-d3
5-Methyl-2'-deoxycytidine-d3 is the deuterium labeled Methyl-2'-deoxycytidine .

Cat. No.	Product Name		Classification

5-Hydroxymethyl-2’-deoxycytidine 5hmdC		Nucleosides and their Analogs C
5-Hydroxymethyl-2’-deoxycytidine (5hmdC) is an oxidation derivative of 5-methyl-2'-deoxycytidine (5-mdC) in DNA. 5-Hydroxymethyl-2’-deoxycytidine may serve as a marker of irreversibly damaged cells .

N4-Acetyl-5’-O-(4,4’-dimethoxytrityl)-5-methyl-2’-deoxycytidine		Nucleosides and their Analogs C
N4-Acetyl-5’-O-(4,4’-dimethoxytrityl)-5-methyl-2’-deoxycytidine is a purine nucleoside analog. Purine nucleoside analogs have broad antitumor activity targeting indolent lymphoid malignancies. Anticancer mechanisms in this process rely on inhibition of DNA synthesis, induction of apoptosis, etc .

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